Cardiogen yog tetrapeptide los ntawm Khavinson luv peptide tsev neeg, tshwj xeeb tshaj yog tsom rau lub plawv rau nws cov kev tswj hwm myocardial. Nws cov kab ke yog Ala-Glu-Asp-Arg, thiab nws qhov hnyav molecular yog kwv yees li 489 Da. Nws ntseeg tau tias nkag mus rau hauv cardiomyocyte nucleus, khi rau cov chaw DNA tshwj xeeb, txhim kho cardiomyocyte protein synthesis, inhibit p53-mediated apoptosis, thiab pab cov laus lossis puas cov ntaub so ntswg myocardial "reboot" qee cov txheej txheem kho.
⚠️ Lus Cim Tseem Ceeb: Tsis pom zoo los ntawm FDA / EMA / NMPA. Cov pov thawj feem ntau yog los ntawm Lavxias kev tshawb fawb thiab kev sim ua ntej. Nws yog ib qho tshuaj tshawb fawb hauv chaw kuaj mob lossis ib tus neeg Lavxias - ua tus tswj kev lom neeg thiab tsis tuaj yeem hloov cov tshuaj kho mob plawv.
Core Functions
1. Tiv thaiv cardiomyocytes thiab inhibits apoptosis: Downregulates pro-apoptotic p53 qhia, txo cardiomyocyte necrosis tshwm sim los ntawm ischemia thiab oxidative kev nyuaj siab, ho txo cell poob tom qab myocardial raug mob, thiab alleviates cardiac cov ntaub so ntswg puas los ntawm myocardial infarction thiab hypoxia.
2. Optimizes myocardial contractile muaj nuj nqi: Upregulates cov synthesis ntawm contraction-txog cov proteins xws li myocardial actin thiab myosin, txhim kho plawv contractile tsis muaj zog los ntawm kev laus thiab kab mob, stabilizing myocardial calcium thauj mechanisms, thiab rov ua kom lub cev muaj zog.
3. Anti-fibrosis thiab txo qhov caws pliav hyperplasia: inhibits kev loj hlob ntawm lub plawv fibroblasts, txo cov collagen ntau dhau tom qab raug mob, tiv thaiv myocardial hardening thiab ventricular remodeling, thiab txo cov kev pheej hmoo ntawm lub plawv tsis ua hauj lwm.
4. Antioxidant thiab mitochondrial tiv thaiv: Ua kom muaj kev ua haujlwm ntawm SOD thiab glutathione antioxidant enzymes hauv myocardium, tshem tawm cov dawb radicals hauv plawv, thiab txo cov kev laus ntawm myocardial thiab tsis muaj zog metabolism tshwm sim los ntawm lub sijhawm ntev oxidation.
5. Kev tswj hwm lub plawv mob me me: Txo qhov kev tso tawm ntawm pro- cov kab mob inflammatory xws li TNF- thiab IL-6 hauv myocardium, alleviating persistent inflammatory puas los ntawm mob myocarditis thiab ischemia-reperfusion.
Core Zoo
1. Kev tsom mus rau myocardium nrog qhov tshwj xeeb siab heev: Lub hom phiaj tsuas yog cov kab mob plawv nkaus xwb, uas yuav luag tsis cuam tshuam rau lwm yam kabmob xws li lub ntsws, lub siab, lub raum, thiab lub paj hlwb. Cov ntsuas ntsuas tau huv, thiab tsis muaj qhov hloov pauv hauv cov ntaub ntawv los ntawm txoj hauv kev tsis sib xws.
2. Cim Nuclear-Level Regulatory Mechanism: Tsis zoo li cov peptide membrane receptors zoo tib yam, nws ncaj qha khi rau myocardial chromatin los qhib cov noob caj noob ces, muab cov ntsiab lus ua tiav ntau dua rau kev tiv thaiv kev laus thiab kho myocardial, ua rau kev tshawb fawb ntau dua.
3. Cov Molecule me me nrog cov dej zoo solubility thiab qis dua -Kev Siv Tau Zoo: Cov tetrapeptide muaj qhov hnyav me me, tso cai rau kev ua haujlwm siab hauv kev nkag mus rau myocardial cell membranes thiab nuclear membranes. Nws yuav tsum tau siv tsawg dua, ua kom ntev -kev sim tsiaj cov nqi- siv tau. Nws yaj yooj yim hauv cov dej ntshiab thiab nruab nrab PBS, tsis tas yuav muaj cov tshuaj solubilizer tshwj xeeb.
4. Kho + Anti-aging + Anti-fibrosis Trinity: Ib qho reagent ib txhij hais txog peb qhov teeb meem loj: myocardial necrosis, txo qis kev ua haujlwm plawv, thiab caws pliav, tshem tawm qhov xav tau ntau lub plawv reagents thiab ua kom yooj yim rau kev sim cov txheej txheem.
5. Mature Research System: Ntau xyoo ntawm cov ntaub ntawv preclinical tsub zuj zuj nyob rau hauv Russia muab mature dosage references rau cov laus lub plawv tsis ua hauj lwm thiab ischemic raug mob qauv, kom muaj kev ruaj ntseg siab nyob rau hauv rov thwmsim.
Cia Lub Sijhawm: Cov khoom no yog qhov muag heev thiab yuav tsum tau ua tib zoo saib xyuas. Yog tias nyob rau hauv daim ntawv qhuav qhuav, nws yuav tsum tau muab kaw kom nruj thiab khaws cia hauv lub tub yees ntawm -20℃kom ruaj khov rau ntau xyoo. Thaum yaj mus rau hauv cov kua, nws yuav tsum tau aliquoted thiab khaws cia ntawm -80 degree. Tsis txhob, tsis txhob khov thiab thaw ntau zaus! Siv tsuas yog cov nyiaj uas xav tau txhua zaus, raws li rov ua kom khov thiab thawing yuav txo nws txoj haujlwm, uas yuav yog qhov pov tseg loj.
Ceev faj & Contraindications
Tsis yog-tshuaj: Cov hmoov lyophilized yog rau kev tshawb fawb hauv chav kuaj xwb; Lavxias teb sab capsules yog cov tshuaj lom neeg regulator / noj tshuaj ntxiv. Tsis txhob tso tseg cov tshuaj kws kho mob raws li lub ntsiab lus ntawm "kev kho mob plawv."
Mob plawv mob hnyav yuav tsum tau saib xyuas kev kho mob: Mob myocardial infarction, tsis ruaj khov angina, NYHA chav kawm III-IV lub plawv tsis ua hauj lwm, mob arrhythmias loj heev- nrhiav kev kho mob hauv tsev kho mob ua ntej; tsis txhob sim "tus kheej- kho" nrog qhov no.
Kev txhawj xeeb txog mob qog noj ntshav: Txoj kev xav, nws tuaj yeem txo qis cov cim apoptosis. Tsis pom zoo rau cov neeg mob uas muaj cov qog nqaij hlav qog nqaij hlav (tshwj xeeb tshaj yog hematologic / cov qog nqaij hlav) lossis cov kws kho mob hauv xov tooj cua lossis kws khomob.
Kev sib cuam tshuam ntawm cov tshuaj hauv plawv: Tam sim no siv -blockers, CCBs, digitalis, anticoagulants, thiab lwm yam.-tsis muaj cov ntaub ntawv qhia meej txog kev sib cuam tshuam; kev siv ua ke yuav tsum tau soj ntsuam los ntawm tus kws kho plawv.
Txhaj tshuaj tiv thaiv: Subcutaneous / intramuscular txhaj tej zaum yuav ua rau lub zos redness, o, me ntsis stinging, los yog induration, uas feem ntau yog nyob rau hauv 24 teev; tig qhov chaw txhaj tshuaj.
Contraindications: Tsis muaj cov ntaub ntawv kev nyab xeeb muaj rau cov poj niam cev xeeb tub, niam pub niam mis, thiab cov neeg muaj hnub nyoog qis dua 18 xyoo; contraindicated nyob rau hauv cov neeg uas muaj hypersensitivity rau peptide Cheebtsam; ceev faj yuav tsum tau siv rau cov neeg uas muaj kab mob autoimmune hnyav.
Tej yam tshwm sim tshwm sim: Qee zaus, qhov chaw txhaj tshuaj tsis xis nyob yuav tshwm sim; daim tawv nqaij pob khaus / khaus yog qhov tsawg heev thiab feem ntau me me thiab tus kheej - txwv.
Yuav Order li cas?
Ua ntej, thov qhia peb txog koj qhov kev xav tau lossis daim ntawv thov scenario.
Thib ob, peb yuav muab cov lus hais raws li koj xav tau lossis peb cov lus qhia.
Thib peb, tus neeg siv khoom lees paub cov qauv thiab them tus nqi tso nyiaj.
Plaub, peb muaj lub luag haujlwm rau kev npaj ntau lawm. Cov luv nqi muaj rau kev txiav txim ntau.
Koj cov khoom zoo li cas? Ua ntej, peb cov khoom zoo heev thiab ruaj khov zoo. Peb ua 100% kuaj ua ntej xa zaub mov.
Yog li ntawd, peb ua nruj tswj xyuas ua ntej ua zaub mov xa tuaj. Yog tias muaj teeb meem zoo tau pom los ntawm peb, peb yuav hloov cov khoom dawb los yog muab cov nyiaj rov qab.
Yuav ua li cas thiaj tsim tau lub sijhawm ntev -, kev koom tes zoo nrog peb?
1. Peb hais kom muab siab - cov khoom zoo thiab cov nqi sib tw kom ntseeg tau cov neeg siv khoom zoo;
2. Peb hwm txhua tus neeg siv khoom, saib lawv li phooj ywg, thiab ua siab dawb ua lag luam thiab ua phooj ywg nrog lawv, txawm lawv tuaj qhov twg los.
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